feedback pathways that report an excess accumulation of ammonia

Both of these mechanisms are reversible inhibitory processes.

A pharmaceutical composition comprising a conjugate of a cytokine and a tumor targeting moiety (TTM) and a pharmaceutically acceptable excipient, wherein the cytokine is present in an amount which does not induce a negative feedback mechanism.

Reverse T3 exerts its negative feedback on T3 and ties up thyroid receptors.

T3 is the metabolically active thyroid hormone, which causes feedback inhibition, and lowers elevated TSH levels.

T3 is the metabolically active thyroid hormone, which causes feedback inhibition and lowers elevated TSH level.

In fact, reverse T3 creates more powerful negative feedback on the production of T3 than vice versa.

At large doses, spermatogenesis may be suppressed through feedback inhibition of pituitary follicle-stimulating hormone(FSH).

At large doses, spermatogenesis may be reduced through feedback inhibition of pituitary follicle-stimulating hormone (FSH).

With large doses of exogenous androgens, spermatogenesis also may be suppressed through feedback inhibition of pituitary follicle-stimulating hormone (FSH) secretion.

With large doses of exogenous androgens, spermatogenesis may also be suppressed through inhibition of pituitary follicle stimulating hormone (FSH).

With large doses of exogenous androgens, spermatogenesis may also be suppressed through inhibition of pituitary follicle stimulating hormone (FSH).

With large doses of exogenous androgens, spermatogenesis also may be suppressed through feedback inhibition of pituitary follicle-stimulating hormone (FSH) secretion.

With large doses of exogenous androgens, spermatogenesis also may be suppressed through feedback inhibition of pituitary follicle-stimulating hormone (FSH) secretion.

Glutathione is a tightly regulated intracellular constituent and is limited in its production by negative feedback inhibition of its own synthesis through the enzyme gamma-glutamylcysteine synthetase.