The present invention provides nucleic acids that include a promoter that is inducible by a transcriptional activator protein; and a nucleotide sequence that encodes the transcriptional activator protein.
The method utilizes a genetically modified tissue plasminogen activator protein as a substrate.
The present invention further disclose the use of the polynucleotide encoding the new Ras GTP activator protein 12.
Prokaryotic tyrosinases that require a separate activator protein, particularly tyrosinases derived from Streptomyces are preferred.
The invention features a novel gene, CRX, which encodes a retinal-specific transcriptional activator protein.
From that point of view, the protein of the present invention is called platelet promoting protein (PPP).
It relates to a method for diagnosis of colorectal cancer from a stool sample, derived from an individual by measuring PSA3 in said sample.
It discloses the use of protein proteasome subunit alpha 3 (PSA3) in the diagnosis of colorectal cancer.
The present invention relates to the diagnosis of colorectal cancer.
Novel single nucleotide polymorphisms in the human Ran GTPase activating protein 1 (RANGAP1) gene are described.
The invention relates to a cell line expressing mutant protein -human tissue-type- plasminogen activator, and the method for constructing it, and to the method for producing the mutant protein.
The novel antitumor agent comprises, as the active ingredient, a substance capable of inhibiting a cell cycle-dependent Rho GTPase activating protein (RhoGAP).
The present invention furthermore relates to an improved method for isolating sRNAs from a sample using HC-Pro together with a binding enhancer protein.
This method for screening a novel antitumor agent is characterized in that a substance capable of inhibiting a cell cycle-dependent Rho GTPase activating protein is selected thereby.
Ras and Rap are regulated by different sets of guanine nucleotide exchange factors and GTPase-activating proteins, thus providing one level of specificity. (wikipedia.org)
The second control mechanism is a response to glucose, which uses the catabolite activator protein (CAP) homodimer to greatly increase production of β-galactosidase in the absence of glucose.
Rarely, infantile Krabbe disease is caused by a mutation in the prosaposin (PSAP) gene (10q21-q22), encoding sphingolipid activator protein saposin-A, necessary for GALC activity.
The methods include identifying therapeutic agents which modulate a CRAC channel and/or a glycoprotein activator of a CRAC channel.
Rarely, infantile Krabbe disease is caused by a mutation in the prosaposin (PSAP) gene (10q21-q22), encoding sphingolipid activator protein saposin-A, necessary for GALC activity.
Also provided are diagnostic methods that ultilise a CRAC channel and/or a glycoprotein activator of a CRAC channel.
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