This is commonly referred to as post-edited machine translation, or PEMT.
Some companies are investing in Post-Edit Machine Translation, or PEMT.
PEMT catalyses methylation of PE, which also produces PC.
This invention is further directed to methods for identifying individuals having or at risk of having PEMT-associated liver disorders.
Post-edited Machine Translation, consisting in reviewing and correcting the document translation generated by the machine translation system.
The glycerolipids are produced through the modification of a cell so as to increase phosphatidic acid via the inhibition of the phosphatidylethanolamine N-methyltransferase (PEMT) pathway.
This invention also provides pharmaceutical compositions and methods for treating PEMT-associated liver disorders by modulating the phosphatidylethanolamine N-methyltransferase (PEMT) pathway and the phosphocholine cytiylyltransferase (CT) pathway.
The present invention provides metabolic markers and targets, especially PEMT activity and metabolic markers and components associated therewith useful for diagnosing and treating various disease conditions.
Post Editing Machine Translation (or PEMT) combines using a translation memory in real time, machine translation updates and human checks.
Active PEMT (n = 17) or placebo treatment (n = 19) was performed three times a week for 3 weeks.
Finally, the invention relates to methods for determining the response of a patient who suffers from cancer to ChoKα-inhibiting agents based on the determination of the PEMT and/or ChoKβ expression levels.
PEMT catalyses methylation of phosphatidylethanolamine (PE) to phosphatidylcholine (PC) in liver cells using S-adenosyl methionine as a methyl donor.
Wada and colleagues fed mice lacking the PEMT enzyme a high-fat high-sucrose diet for up to 90 weeks.
Therefore, it can be concluded that variations in PEMT activity and individual choline requirements can determine whether someone is able to get sufficient choline from a vegan diet.
The researchers noted enhanced apoptosis and cell proliferation in the liver, which they explain through transactivation of the protein p53, which is inhibited in the presence of PEMT.
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