Beta-glucuronidase is an enzyme that helps to break down large molecules.
Calcium-d-glucarate inhibits beta-glucuronidase which allows toxins to be removed.
Reducing beta-glucuronidase enhances enterohepatic (liver) circulation which reduces cholesterol synthesis.
The present invention relates to a method for de-glycosylation of baicalin to produce baicalein by treating the baicalin with an enzyme having beta-glucuronidase activity under conditions such that baicalin is converted to baicalein.
Low levels of CDG are correlated with higher levels of beta-glucuronidase.
In addition, uses of the β-glucuronidase as a visual and as a selectable marker for transformation are also described.
Gene products, expression vectors and host cells suitable for expressing β-glucuronidase are also provided.
This important detoxification process can be disrupted by the enzyme betaglucuronidase, which acts to reactivate the neutralized toxins.
At the time of Project 259, a link between beta-glucuronidase and bladder cancer had already been implied.
Trenbolone and it’s derivative 17epi-trenbolone are both excreted in urine as conjugates that can be hydrolyzed with beta-glucuronidase.
Trenbolone and 17epi-trenbolone are both excreted in urine as conjugates that can be hydrolyzed with beta-glucuronidase.
An EPD shot contains a very minute amount of many allergens plus an enzyme which naturally occurs in the human body, beta-glucuronidase.
Preliminary studies in humans show that D-Glucarate supplements are safe, and have the same effects on beta-glucuronidase in people that they do in animals.
T-DNA tagging with a promoterless β-glucuronidase (GUS) gene generated transgenic Nicotiana tabacum plants that expressed GUS activity either only in developing seed coats, or constitutively.
Preliminary studies in humans show that D-Glucarate supplements are safe, and that they have the same effects on beta-glucuronidase in people that they do in animals.
OPCs are also thought to limit the proteolytic enzymes collagenase, elastase, hyaluronidase and beta glucuronidase involved in cartilage and vasculature degeneration.
It is caused by a deficiency of beta-glucuronidase.
Bacterial enzymes like beta-glucuronidase hydrolyze conjugated estrogens and bile acids.
Beta-glucuronidase and other hydrolytic bacterial enzymes also deconjugate bile acids.
The bacterial beta-glucuronidase inhibitors are selective for the inhibition of bacterial beta-glucuronidase enzymes and do not inhibit mammalian beta-glucuronidase.
Beta-glucuronidase and other hydrolytic bacterial enzymes also deconjugate bile acids.
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